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Cardiovascular Research Advance Access first published online on June 25, 2008
This version [Accepted Manuscript] published online on July 9, 2008
Cardiovascular Research, doi:10.1093/cvr/cvn174
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org

Vascular tolerance to nitroglycerin in ascorbate deficiency: results are in favour of an important role of oxidative stress in nitrate tolerance

Andreas Daiber

II. Medizinische Klinik – Labor für
Molekulare Kadiologie
Klinikum der Johannes
Gutenberg-Universität
Verfügungsgebäude für Forschung
und Entwicklung
Raum 00349
Obere Zahlbacher Str. 63
55101 Mainz
Germany
Tel: +49 6131 33301
Fax: +49 6131 33304
E-mail address: daiber@uni-mainz.de

Tommaso Gori

II. Medizinische Klinik – Labor für
Molekulare Kadiologie
Klinikum der Johannes
Gutenberg-Universität
Mainz
Germany

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In their recent article ‘Vascular tolerance to nitroglycerin (GTN) in ascorbate deficiency' Wölkart et al. demonstrated an impaired vasodilator potency of GTN in ascorbate-deficient guinea pigs.1 The authors also conclude from the lack of an effect of polyethylene-glycolated superoxide dismutase (PEG-SOD) and catalase (PEG-catalase), that the impairment of GTN vasodilator potency induced by ascorbate deficiency is not mediated by induction of oxidative stress (their Figure 3). Although these data are interesting, especially in . . . [Full Text of this Article]


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Vascular tolerance to nitroglycerin in ascorbate deficiency: results are in favour of an important role of oxidative stress in nitrate tolerance: reply
Cardiovasc Res, September 1, 2008; 79(4): 724 - 724.
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