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Cardiovascular Research Advance Access first published online on April 25, 2008
This version [Corrected Proof] published online on May 14, 2008
Cardiovascular Research, doi:10.1093/cvr/cvn103
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org

A new favourable effect of cocoa on atherosclerosis?

José A. Páramo*

Hematology Service, University Clinic, Divison of Cardiovascular Sciences, Centre for Applied Medical Research, University of Navarra, Avda PIO XII-55, 31008 Pamplona, Spain

* Corresponding author: Tel: +34 948296397; fax: +34 948296500. E-mail address: japaramo@unav.es

This editorial refers to ‘Cocoa procyanidins inhibit expression and activation of MMP-2 in vascular smooth muscle cells by direct inhibition of MEK and MT1-MMP activities’ by K.W. Lee et al., doi:10.1093/cvr/cvn056.

The first 10% of the full text of this article appears below.

Numerous epidemiological studies have indicated a role of different nutrients and phytochemicals in reducing the risk of cardiovascular disease, although the mechanisms are far from clear. Cocoa is one of the most concentrated sources of flavonols, a subgroup of the natural antioxidant compounds called flavonoids, also found in tea and red wine. Flavonoids, in turn, are a subgroup of compounds called polyphenols. Animal as well as human studies have shown beneficial changes in biological phenomena related to cardiovascular health after the consumption of polyphenol-rich foods such as cocoa, red wine, tea, and berries.1–3 Evidence that flavonol-rich cocoa . . . [Full Text of this Article]


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Related articles in Cardiovasc Res:

Cocoa procyanidins inhibit expression and activation of MMP-2 in vascular smooth muscle cells by direct inhibition of MEK and MT1-MMP activities
Ki Won Lee, Nam Joo Kang, Min-Ho Oak, Mun Kyung Hwang, Jong Hun Kim, Valérie B. Schini-Kerth, and Hyong Joo Lee
Cardiovasc Res 2008 79: 34-41. [Abstract] [FREE Full Text]  

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Cocoa procyanidins inhibit expression and activation of MMP-2 in vascular smooth muscle cells by direct inhibition of MEK and MT1-MMP activities
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