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Cardiovascular Research Advance Access originally published online on October 1, 2008
Cardiovascular Research 2008 80(3):326-327; doi:10.1093/cvr/cvn272
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org

Pulse pressure to vascular cells: novel methods for solving pathogenesis in cell migration

Yukisato Ishida*

Laboratory of Physiology and Pharmacology, Faculty of Health Science Technologies, Bunkyo Gakuin University, 1196 Kamekubo, Fujimino-shi, Saitama 356-8533, Japan

* Corresponding author. Tel: + 81 49 261 7943; fax: + 81 49 261 8923. E-mail address: ishiday@hst.u-bunkyo.ac.jp

This editorial refers to ‘Enhanced pulsatile pressure accelerates vascular smooth muscle migration: implications for atherogenesis of hypertension’ by Tada et al.,2 pp. 346–353, this issue.

The first 10% of the full text of this article appears below.

Vascular smooth muscle migration is a risk factor for developing atherosclerosis, a major cause of morbidity and mortality. A cohort study carried out over 25 years in seven countries revealed that pulse pressure, diastolic and systolic pressures are the best predictors of cardiovascular disease death.1 In this issue of Cardiovascular Research, Tada et al. (Shimokawa's group) describe a novel method using a . . . [Full Text of this Article]


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Enhanced pulsatile pressure accelerates vascular smooth muscle migration: implications for atherogenesis of hypertension
Tomohiro Tada, Jun Nawata, Huan Wang, Noriko Onoue, Doe Zhulanqiqige, Kenta Ito, Koichiro Sugimura, Yoshihiro Fukumoto, and Hiroaki Shimokawa
Cardiovasc Res 2008 80: 346-353. [Abstract] [Full Text] [PDF]