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Cardiovascular Research Advance Access originally published online on February 5, 2008
Cardiovascular Research 2008 78(2):242-249; doi:10.1093/cvr/cvn027
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please email: journals.permissions@oxfordjournals.org

Post-ischaemic neovascularization and inflammation

Jean-Sebastien Silvestre*, Ziad Mallat, Alain Tedgui and Bernard I. Lévy

Cardiovascular Research Center, Inserm Lariboisiere, Inserm U689, Universite Paris 7, Hôpital Lariboisière, 41 Bd de la Chapelle, 75475 Paris cedex 10, France

* Corresponding author. Tel: +33 153216702; fax: +33 142813128. E-mail address: jean-sebastien.silvestre{at}larib.inserm.fr

Four principal processes—vasculogenesis, angiogenesis, arteriogenesis, and collateral growth—characterize tissue repair and remodelling in acute and chronic ischaemic vascular diseases. The relative importance of each process remains unclear, but it is likely that they may complement each other. In addition, these processes are driven by distinct, but partially overlapping, cellular and molecular pathways. In particular, inflammation might be one of the most important stimuli for initiation of vessel growth in the setting of ischaemia. Here, we summarize the current knowledge on the inflammatory response in the context of ischaemia and review the major factors that may be involved in inflammation-induced, post-ischaemic neovascularization.

KEYWORDS Inflammation; Ischaemia; Angiogenesis; Arteriogenesis

Time for primary review: 22 days


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