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Cardiovascular Research Advance Access originally published online on January 14, 2008
Cardiovascular Research 2008 77(4):612-613; doi:10.1093/cvr/cvn010
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2008. For permissions please e-mail: journals.permissions@oxfordjournals.org

Stem cell mobilization versus stem cell homing: potential role for parathyroid hormone?

Klaus-Dieter Schlüter*, Rolf Schreckenberg and Sibylle Wenzel

Physiologisches Institut, Aulweg 129, D-35392 Giessen, Germany

* Corresponding author. Tel. +49 641 99 47222; fax +49 641 99 47239. E-mail address: klaus-dieter.schlueter@physiologie.med.uni-giessen.de

See article by Zaruba et al.,1 pp. 722–731.

The first 10% of the full text of this article appears below.

Stem cells are considered as potential candidates to regenerate the injured myocardium. It has been shown that stem cells are able to differentiate into cardiomyocytes, as recently reviewed.2 It is still an open question, however, whether such myocytes contribute to the functional recovery of the post-infarcted heart. This process is limited by the small number of cells that differentiate into cardiomyocytes, the limited coupling of pre-existing cardiomyocytes to these newly formed myocytes, and the question whether the overall increases in ejection fractions indeed depend on the amount of cardiomyocytes newly formed by stem cells. A specific pattern of matrix proteins may be expressed by these . . . [Full Text of this Article]


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Parathyroid hormone treatment after myocardial infarction promotes cardiac repair by enhanced neovascularization and cell survival
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Cardiovasc Res 2008 77: 722-731. [Abstract] [FREE Full Text]  

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