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Cardiovascular Research 2007 73(3):448-449; doi:10.1016/j.cardiores.2006.11.014
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Copyright © 2006, European Society of Cardiology

Protecting endothelial function: A novel therapeutic target of ATP-sensitive potassium channel openers

Tetsuo Minamino and Masatsugu Hori*

Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine, Suita, Osaka 565-0871, Japan

* Corresponding author. Tel.: +81 6 6879 3635; fax: +81 6 6879 3634. Email address: mhori@medone.med.osaka-u.ac.jp

Received 5 November 2006; accepted 13 November 2006

The first 10% of the full text of this article appears below.

See article by Wang et al. [7] (pages 497–503) in this issue.

Endothelial dysfunction is commonly observed in patients with hypertension, type II diabetes mellitus, and hypercholesteremia and is considered to be an early event in the changes accompanying atherosclerosis [1,2]. Disruption of normal endothelial function leads to loss of vasomotor control, reduced production of nitric oxide (NO), formation of a procoagulant surface, and promotion of inflammation [1,2]. These events may lead to destabilization of atherosclerotic plaques and could cause acute coronary syndrome. Indeed, recent clinical studies . . . [Full Text of this Article]


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