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Cardiovascular Research 2007 73(1):6-7; doi:10.1016/j.cardiores.2006.11.013
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Copyright © 2006, European Society of Cardiology

Estrogen protection, oxidized LDL, endothelial dysfunction and vasorelaxation in cardiovascular disease: New insights into a complex issue

C. Subah Packer*

Indiana University School of Medicine, Department of Cellular and Integrative Physiology, 635 Barnhill Drive, Indianapolis, Indiana 46202-5120, United States

* Tel.: +1 317 274 9765; fax: +1 317 274 3318. Email address: spacker1@iupui.edu

Received 5 November 2006; accepted 9 November 2006

The first 10% of the full text of this article appears below.

See article by Monsalve et al. [14] (pages 66–72) in this issue.

Pre-menopausal women have a lower risk of developing hypertension than men or post-menopausal women [1]. Estrogens may prevent or delay the onset of hypertension in pre-menopausal women. Similarly, animal models such as the spontaneously hypertensive rat (SHR) exhibit sexual dichotomy in development of essential hypertension, and estrogen reduces blood pressure in hypertensive female rats [2,3]. Such animal models are useful in the study of the protective effects of estrogen in the development . . . [Full Text of this Article]


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