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Cardiovascular Research 2006 72(2):196-197; doi:10.1016/j.cardiores.2006.08.015
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Copyright © 2006, European Society of Cardiology

Gap junction heterogeneity in reentrant ventricular tachycardia

Richard D. Veenstra*

SUNY Upstate Medical University, Department of Pharmacology, 750 East Adams Street Syracuse, NY 13210, United States

* Corresponding author. Tel.: +1 315 464 5145; fax: +1 315 464 8014. Email address: veenstrr@upstate.edu

Received 19 August 2006; accepted 24 August 2006

The first 10% of the full text of this article appears below.

See article by Cabo et al. [18] (pages 241–249) in this issue.

The cells of all tissues are coupled by gap junctions, with the exception of blood and adult skeletal muscle cells. Gap junctions provide for homoeostasis and integrative function by mediating the exchange of ions and chemical second messenger molecules between adjacent cells. The electrical signaling provided by these specialized intercellular junctions is perhaps best understood in the heart, where their abundance serves to provide low electrical resistance pathways for current flow – ensuring fast and efficient transfer of . . . [Full Text of this Article]


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