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Cardiovascular Research 2006 72(1):5-6; doi:10.1016/j.cardiores.2006.07.021
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Copyright © 2006, European Society of Cardiology

Atrial gap junction remodeling: Looking for lost gaps and orphaned connexins in three dimensions

René J.P. Musters*

Laboratory for Physiology (ICaR-VU), VU University Medical Center (VUMC), Van der Boechorststraat 7, 1081 BT Amsterdam, The Netherlands

* Tel.: +31 20 444 1749; fax: +31 20 444 8255. Email address: r.musters@vumc.nl

Received 19 July 2006; accepted 25 July 2006

The first 10% of the full text of this article appears below.

See article by Rucker-Martin et al. [22] (pages 69–79) in this issue.


   1. Connexins and gap junction channel function in the heart
 
Gap junction channels are composed of members of a multi-gene family of proteins called connexins. These proteins are named by the abbreviation Cx followed by the molecular weight of the specific protein. Mammalian cardiac myocytes express Cx43, Cx45, and Cx40, the former being the major cardiac gap junction protein, which is expressed in atrial and ventricular myocytes as well as in selected regions of the atrioventricular conduction system [1,2]. Individual gap junction channels (also called connexons) formed by multiple connexin . . . [Full Text of this Article]


   2. Gap junction remodeling and electrical uncoupling in myocardial ischemia, cardiac hypertrophy and heart failure
 

   3. Cardiac gap junction remodeling and lateral redistribution of connexins
 

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