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Cardiovascular Research 2005 68(3):502-503; doi:10.1016/j.cardiores.2005.07.020
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Copyright © 2005, European Society of Cardiology

The electrophysiological basis of MAP recordings

Edward J. Vigmond*

Department of Electrical and Computer Engineering, University of Calgary, 2500 University Dr. NW, Calgary, Alberta, Canada T2N 1N4

* Tel.: +1 403 210 3887; fax: +1 403 282 6855. Email address: vigmond@ucalgary.ca

Received 22 July 2005;
The first 10% of the full text of this article appears below.

Recently, differing views as to the genesis of monophasic action potential (MAP) recordings have surfaced [1–3]. Kondo et al. [1,3] claim that MAP recordings come about because core conductor theory dictates that the extracellular potential is a scaled version of the transmembrane voltage and that the contact electrode acts as a stationary ground. Franz [2] holds to the view that injury currents at the boundary of the contact electrode zone of influence lead to extracellular potential changes which reflect changes in transmembrane voltage. As a consequence of these divergent views, the importance of one electrode with respect to the other has come into question.

MAP electrodes are similar to potentials recorded with . . . [Full Text of this Article]


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