Copyright © 2005, European Society of Cardiology
Transmural dispersion in LQT3 and arrhythmogenesis
Hospital of the Westfälische Wilhelms-University, Department of Cardiology and Angiology, Münster, Germany
* Corresponding author. Medizinische Klinik und Poliklinik C, -Kardiologie und Angiologie-, Universitätsklinikum Münster, Albert-Schweitzer Str. 33, D-48149 Münster. Tel.: +49 251 834 5160; fax: +49 251 834 9943. Email address: milbergp@uni-muenster.de
| The first 10% of the full text of this article appears below. |
We thank Dr. Opthof and Dr. Coronel for the interest in our recently published work [1] and appreciate their comments. We aimed at developing a novel intact heart model of LQT3 for a better understanding of the mechanisms by which functional electrical instability at the level of the whole
![]()
CiteULike
Connotea
Del.icio.us What's this?
This article has been cited by other articles:
![]() |
G. Thomas, I. S. Gurung, M. J. Killeen, P. Hakim, C. A. Goddard, M. P. Mahaut-Smith, W. H. Colledge, A. A. Grace, and C. L.-H. Huang Effects of L-type Ca2+ channel antagonism on ventricular arrhythmogenesis in murine hearts containing a modification in the Scn5a gene modelling human long QT syndrome 3 J. Physiol., January 1, 2007; 578(1): 85 - 97. [Abstract] [Full Text] [PDF] |
||||
