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Cardiovascular Research 2005 67(4):581-582; doi:10.1016/j.cardiores.2005.06.019
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Copyright © 2005, European Society of Cardiology

SERCA upregulation: Breaking the positive feedback in heart failure?

José W.M. Bassani* and Rosana A. Bassani

Centro de Engenharia Biomédica, Universidade Estadual de Campinas, Campinas SP, Brazil
Department, Engenharia Biomédica/FEEC, Universidade Estadual de Campinas, Campinas SP, Brazil

* Corresponding author. Centro de Engenharia Biomédica/UNICAMP, Caixa Postal 6040, 13084-971 Campinas, SP, Brazil. Tel.: +55 19 3788 9274; fax: +55 19 3289 3346. Email address: bassani@ceb.unicamp.br

Received 17 June 2005; revised 21 June 2005;
The first 10% of the full text of this article appears below.

See article by Maier et al. [9] (pages 636–646) in this issue.

Much effort has been devoted to devising approaches that may improve survival and the quality of life for the millions of people afflicted by heart failure (HF) all over the world. Because HF develops in association with different etiologies, its treatment represents a challenge for clinicians and investigators. The consistent observation of defective Ca2+ cycling in the failing myocardium has led to the notion that improvement of cardiac Ca2+ handling might be a suitable approach to minimize the systolic and diastolic dysfunction and possibly also the electrophysiological abnormalities that . . . [Full Text of this Article]


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