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Cardiovascular Research 2005 67(1):9-10; doi:10.1016/j.cardiores.2005.05.007
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Copyright © 2005, European Society of Cardiology

Stretch-stimulated ANF secretion: Are the ATP-sensitive potassium channels the missing link?

Liliana Graciela Bianciotti*

Cátedra de Fisiopatología, Facultad de Farmacia y Bioquímica, Universidad de Buenos Aires. Junin 956 Piso 5, Buenos Aires, 1113AAD, Argentina

* Tel.: +54 11 4964 8268x37. Email address: lbianc@ffyb.uba.ar

Received 20 April 2005; accepted 3 May 2005

The first 10% of the full text of this article appears below.

See article by Saegusa et al. [9] (pages 60–68) in this issue.

In 1981, de Bold et al. reported that the administration of atrial extracts to rats induced diuresis and natriuresis [1]. Soon afterwards, the substance with diuretic and natriuretic properties present in the atrial extracts was identified as the atrial natriuretic peptide (ANF) [2]. This important discovery led to the new concept that the heart was not only a mechanical pump but also an endocrine organ. Since the characterization of ANF, many reports appeared in the literature describing a wide spectrum of biological actions displayed by this peptide supported by the wide distribution of the receptors in . . . [Full Text of this Article]


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