Skip Navigation

Cardiovascular Research 2004 64(2):195-197; doi:10.1016/j.cardiores.2004.08.011
© 2004 by European Society of Cardiology
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Kohl, P.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Kohl, P.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2004, European Society of Cardiology

Cardiac cellular heterogeneity and remodelling

Peter Kohl*

Laboratory of Physiology, University of Oxford, Parks Road, OX1 3PT, Oxford, United Kingdom

* Tel.: +44 1865 272 114; fax: +44 1865 272 554. Email address: peter.kohl@physiol.ox.ac.uk

Received 16 August 2004; accepted 18 August 2004

The first 10% of the full text of this article appears below.

See article by Zorn-Pauly et al. [17] (pages 250–259) in this issue.

Uniform cardiac pump function requires well-coordinated myocardial heterogeneity at the cellular level. This insight was eloquently presented some 15 years ago by Katz and Katz [9], who likened the serial and parallel summation of mechanical activity in various regions of cardiac muscle to the combined action of oarsmen on ancient Greek triremes. These battleships were powered by 170 men, placed in three tiers along both sides of the ship. In order to synchronise mechanical action and to achieve optimal propulsion, their rowing gear was ingeniously fine-tuned to match positional requirements . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Circ. Res.Home page
P. Snider, K. N. Standley, J. Wang, M. Azhar, T. Doetschman, and S. J. Conway
Origin of Cardiac Fibroblasts and the Role of Periostin
Circ. Res., November 6, 2009; 105(10): 934 - 947.
[Abstract] [Full Text] [PDF]

Home page
 Am. J. Physiol. Heart Circ. Physiol.Home page
I. Banerjee, J. W. Fuseler, A. R. Intwala, and T. A. Baudino
IL-6 loss causes ventricular dysfunction, fibrosis, reduced capillary density, and dramatically alters the cell populations of the developing and adult heart
Am J Physiol Heart Circ Physiol, May 1, 2009; 296(5): H1694 - H1704.
[Abstract] [Full Text] [PDF]

Home page
 Am. J. Physiol. Heart Circ. Physiol.Home page
I. Banerjee, J. W. Fuseler, R. L. Price, T. K. Borg, and T. A. Baudino
Determination of cell types and numbers during cardiac development in the neonatal and adult rat and mouse
Am J Physiol Heart Circ Physiol, September 1, 2007; 293(3): H1883 - H1891.
[Abstract] [Full Text] [PDF]

Home page
 Circ. Res.Home page
T. K. Borg and R. Markwald
Periostin: More Than Just an Adhesion Molecule
Circ. Res., August 3, 2007; 101(3): 230 - 231.
[Full Text] [PDF]

Home page
 Am. J. Physiol. Heart Circ. Physiol.Home page
T. A. Baudino, W. Carver, W. Giles, and T. K. Borg
Cardiac fibroblasts: friend or foe?
Am J Physiol Heart Circ Physiol, September 1, 2006; 291(3): H1015 - H1026.
[Abstract] [Full Text] [PDF]