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Cardiovascular Research 2004 63(4):580-581; doi:10.1016/j.cardiores.2004.06.027
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

What is a monophasic action potential?

Alan Kadish*

Division of Cardiology, Department of Medicine, The Feinberg School of Medicine, Northwestern University, 251 E. Huron, Feinberg Pavilion, Suite 8-536, Chicago, IL 60611, United States

* Tel.: +1 312 926 0630; fax: +1 312 926 0607. Email address: a-kadish@northwestern.edu

Received 24 June 2004; accepted 28 June 2004

The first 10% of the full text of this article appears below.

See article by Kondo et al. [6] (pages 635–644) in this issue.

One approach to developing better pharmacologic therapy for arrhythmias is to define the electrophysiologic mechanism for clinical rhythm disturbances. However, this approach is problematic, because extracellular electrograms do not provide precise information regarding the dispersion of repolarization or after-depolarizations. Floating microelectrodes have limitations in an experimental laboratory and are not widely applicable clinically. Thus, the ability to record monophasic action potentials (MAPs) using contact catheter electrodes in both experimental and clinical situations has raised the hope that cellular properties that are important in arrhythmogenesis can be . . . [Full Text of this Article]


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V. V. Nesterenko, M. Kondo, and C. Antzelevitch
Biophysical basis for monophasic action potential
Cardiovasc Res, March 1, 2005; 65(4): 942 - 944.
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