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Cardiovascular Research 2004 63(2):194-195; doi:10.1016/j.cardiores.2004.05.005
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

Tachycardia-induced remodeling: atria and ventricles take a different route

Andreas Goette*,a and Uwe Lendeckelb

aDepartment of Internal Medicine, University Hospital Magdeburg, Division of Cardiology, Leipziger Str. 44, 39120 Magdeburg, Germany
bInstitute of Experimental Internal Medicine, University Hospital Magdeburg, Magdeburg, Germany

* Corresponding author. Tel.: +49-391-6713203; fax: +49-391-6713202. Email address: andreas.goette@medizin.uni-magdeburg.de

Received 23 April 2004; accepted 10 May 2004

The first 10% of the full text of this article appears below.

See article by Hanna et al. [7] (pages 236–244) in this issue.

Myocardial remodeling is a common cardiac response to various pathological stimuli and may refer to changes in structure and function (structural remodeling) or electrophysiologic properties (electrical or ionic remodeling) [1–3]. Such disease-induced changes of tissue composition and architecture or function occur in both the atrium and/or the ventricle, and appear to progress steadily in a vicious cycle that, if not halted, eventually causes death of the patient [1]. Structural remodeling itself has a substantial influence on cardiac conduction properties [4]. In particular, the development of conduction inhomogeneities, including areas of slow conduction and conduction . . . [Full Text of this Article]


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