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Cardiovascular Research 2004 62(1):7-8; doi:10.1016/j.cardiores.2004.01.034
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

Angiotensin AT2 receptor: the younger sibling attracts attention

Elisabetta Cerbai and Alessandro Mugelli*

Department of Preclinical and Clinical Pharmacology, University of Firenze, Viale Pieraccini 6, Florence 50139, Italy

* Corresponding author. Tel.: +39-554271264; fax: +39-554271285. Email address: alessandro.mugelli@unifi.it

Received 26 January 2004; accepted 29 January 2004

The first 10% of the full text of this article appears below.

See article by Caballero et al. [6] (pages 86–95) in this issue.

Electrical remodeling is a crucial aspect of the general process of cardiac remodeling associated with left ventricular hypertrophy and heart failure [1], and it is thought to contribute to an increased risk of ventricular fibrillation and sudden cardiac death. Angiotensin II has long been recognized as a major factor in cardiac remodeling in a variety of cardiovascular and non-cardiovascular diseases—from hypertension to diabetes [2]. Two receptor subtypes for angiotensin II have been identified in the heart, AT1 and AT2, which apparently share two features: the endogenous ligand and the gross molecular structure, belonging . . . [Full Text of this Article]


   1. Angiotensin receptors and electrophysiological remodeling
 

   2. Physiological and clinical implications and open questions
 

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