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Cardiovascular Research 2004 61(4):648-650; doi:10.1016/j.cardiores.2004.01.003
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

P21Waf1/Cip1 in endothelial cell survival

Fawzi Aoudjit and Jean Sévigny*

Centre de recherche en Rhumatologie et Immunologie, Centre hospitalier de l’Université Laval, 2705 Boulevard Laurier, Room T1-49, Ste.-Foy, Québec, Canada G1V 4G2

* Corresponding author. Tel.: +1-418-656-4141x46319; fax: +1-418-654-2765. jean.sevigny@crchul.ulaval.ca

Received 22 December 2003;
The first 10% of the full text of this article appears below.

See article by Mattiussi et al. [13] (pages 693–704) in this issue.

The role of endothelial cells in pathological processes is the focus of intensive research. In this context, programmed cell death (apoptosis) plays an important function. Although apoptosis can be seen in many instances as detrimental in the cardiovascular system, it also has beneficial functions. For example, the occurrence of endothelial cell apoptosis has deleterious effects on the development of the cardiovascular system that lead to embryonal lethality. An increase of apoptosis in endothelial cells also contributes to various pathological conditions such as atherosclerosis, tissue destruction during vascular injury, inflammation and allograft arteriopathy, and cellular resistance to apoptosis is necessary for . . . [Full Text of this Article]


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