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Cardiovascular Research 2003 59(2):266-267; doi:10.1016/S0008-6363(03)00465-6
© 2003 by European Society of Cardiology
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Copyright © 2003, European Society of Cardiology

Mitochondria as common endpoints in early and late preconditioning

G. Taimor*

Physiologisches Institut, Justus-Liebig-Universität, Aulweg 129, 35392 Gießen, Germany

* Tel.: +49-641-9947-246; fax: +49-641-9947-219. gerhild.taimor@physiologie.med.uni-giessen.de

The first 10% of the full text of this article appears below.

See article by Rajesh et al. [5] (pages 297–307) in this issue.

Preconditioning is a phenomenon in which short periods of non-lethal ischemia protect the heart against subsequent prolonged ischemic periods. This protective effect, caused by ischemic preconditioning (IPC), has two windows: The early window protects the heart against damage caused by an immediately subsequent, prolonged ischemia and lasts up to 2 to 3 hours, . . . [Full Text of this Article]


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