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Cardiovascular Research 2002 55(4):706-707; doi:10.1016/S0008-6363(02)00531-X
© 2002 by European Society of Cardiology
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Copyright © 2002, European Society of Cardiology

Response to: Effect of inhibition of Na+/Ca+ exchanger at the time of myocardial reperfusion on hypercontracture and cell death

Steve Anderson*

Department of Human Physiology, University of California, One Shields Avenue, Davis, CA 95616-8644, USA

* Tel.: +1-530-752-7621; fax: +1-530-752-5423 seanderson@ucdavis.edu

Received 13 June 2002; accepted 17 June 2002

The first 10% of the full text of this article appears below.

See article by Inserte et al. [12] (pages 739–748) in this issue.

The functional and biochemical characterization of the sarcolemmal Na/Ca exchanger (NCX) testifies to the success of novel approaches by numerous investigators [1,2] and, in particular, development of new techniques for measuring sarcolemmal ion fluxes and cytosolic Na and Ca concentrations ([Na]i and [Ca]i) [3,4]. Our current understanding of the role NCX plays in ischemia-induced changes in cytosolic calcium concentration ([Ca]i) is, to a great extent, derived from studies which used recently developed technology to measure intracellular Na and Ca in the . . . [Full Text of this Article]


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