Skip Navigation

Cardiovascular Research 2001 52(1):164-165; doi:10.1016/S0008-6363(01)00414-X
© 2001 by European Society of Cardiology
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Verma, S.
Right arrow Articles by Anderson, T. J
Right arrow Search for Related Content
PubMed
Right arrow Articles by Verma, S.
Right arrow Articles by Anderson, T. J
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2001, European Society of Cardiology

Re: Endothelin antagonism and internal mammary artery endothelial function

[Cardiovascular Research 2001;52:160–162]

Subodh Verma*, Lawrence Ko and Todd J Anderson

Division of Cardiac Surgery, The Toronto General Hospital, EN 14-215, 200 Elizabeth Street, Toronto, Ont., Canada M5G 2C4

* Corresponding author. Tel.: +1-416-693-6226; fax: +1-416-693-2538 subodh.verma@sympatico.ca

Received 18 July 2001; accepted 18 July 2001

The first 10% of the full text of this article appears below.

To the editor,

We thank Glodny and Barton for their interpretation of our paper and the accompanying editorial. A few important issues were raised which we would like to address. We agree with the authors regarding the resilience of the internal mammary artery (IMA) and its intrinsic resistance against atherosclerosis. This has been ascribed to greater endothelium-dependent nitric oxide (NO) production/availability of this conduit. Atherosclerotic vascular disease does occur . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?