Skip Navigation

Cardiovascular Research 2000 45(1):190-193; doi:10.1016/S0008-6363(99)00326-0
© 2000 by European Society of Cardiology
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Janse, M. J
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Janse, M. J
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2000, European Society of Cardiology

ST segment mapping and infarct size

Michiel J Janse*

Department of Clinical and Experimental Cardiology and The Interuniversity Cardiology Institute, Amsterdam, The Netherlands

* Tel.: +31-20-566-3264; fax: +31-20-697-5458 m.kraayenhof@amc.uva.nl

KEYWORDS Antiarrhythmic agents; Autonomic nervous system; ECG; Infarction; Ischemia

The first 150 words of the full text of this article appear below.


   1 Introduction
 
Following the pioneering studies of Julian [1] and Lown and coworkers [2], demonstrating that ventricular fibrillation is common in patients with acute ischaemia, with and without infarction, and that it can be treated, coronary care units were installed in many medical centres, and attempts were made to hospitalise patients with acute ischaemic attacks as early as possible. As Professor Shillingford writes in this issue [3], the introduction of the coronary care unit offered the possibility to study patients who developed a myocardial infarction in a variety of ways. Pelides et al. [4] reported on nine patients ‘who had sustained an acute myocardial infarction within 72 hours’ in whom the administration of the beta-adrenergic blocker practolol reduced precordial ST segment elevation, which was interpreted as a reduction in the extent and severity of ischaemia.

The background for this 1972 study formed experimental studies by Maroko et al. in 1971 . . . [Full Text of this Article]


   2 Changes in intra- and extracellular potentials during acute ischaemia
 

   3 Concluding remarks
 

Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 J CARDIOVASC PHARMACOL THERHome page
D. Brasil, R. M. Temsah, K. Kumar, H. Kumamoto, N. Takeda, and N. S. Dhalla
Blockade of 5-HT2A Receptors by Sarpogrelate Protects the Heart Against Myocardial Infarction in Rats
Journal of Cardiovascular Pharmacology and Therapeutics, March 1, 2002; 7(1): 53 - 59.
[Abstract] [PDF]