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Corrigendum for Tan et al., Cardiovasc Res 57 (4) 961-973.
Cardiovascular Research 2003 59(3):799-802; doi:10.1016/S0008-6363(03)00398-5
© 2003 by European Society of Cardiology
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Copyright © 2003, European Society of Cardiology

Corrigendum to: "Genetic control of sodium channel function"

[Cardiovascular Research 57 (2003) 961–973]{star}

Hanno L Tana,*, Connie R Bezzinaa,b, Jeroen P.P Smitsa, Arie O Verkerka and Arthur A.M Wildea

aExperimental and Molecular Cardiology Group, Department of Cardiology, Academic Medical Center, Room M0-052, P.O. Box 22700, 1100 DE Amsterdam, The Netherlands
bDepartment of Clinical Genetics, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands

h.l.tan{at}amc.uva.nl

* Corresponding author. Tel.: +31-20-566-3265; fax: +31-20-697-5458.

In the original article Figs. 2, 3 and 4GoGo were incorrect. The correct figures are shown on the following pages.


Figure 2
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  		Fig. 2 (A) Representative electrocardiogram of Long QT syndrome type 3 (40 ms/div). Note marked QT interval prolongation with late peaked T waves. There is also sinus bradycardia. (B) SCN5A mutations associated with Long QT syndrome type 3: summary of changes in their biophysical properties. {tau}fast current decay, time constant of fast component of sodium current decay (fast inactivation); inact, inactivation; V1/2 of inactivation, voltage at which 50% of sodium channels are inactivated; V1/2 of activation, voltage at which 50% of sodium channels are activated; –shift, shift to negative voltage; +shift, shift to positive voltage; {downarrow}, reduction; {uparrow}, increase; =, unchanged; –, not reported.

 

Figure 3
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  		Fig. 3 (A) Representative electrocardiogram of Brugada syndrome (40 ms/div). Note ST segment elevation (coved type) with negative T waves, typically seen in right precordial leads V1–V3 (here V1). There is also marked PQ interval prolongation. (B) SCN5A mutations associated with Bruguda syndrome: summary of changes in their biophysical properties. Same abbreviations as in Fig. 2B.

 

Figure 4
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  		Fig. 4 (A) Representative electrocardiogram of isolated conduction disease (40 ms/div). Note marked QRS widening and PQ interval prolongation. (B) SCN5A mutations associated with isolated conduction disease: summary of changes in their biophysical properties. Same abbreviations as in Fig. 2.

 


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This Article
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