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Cardiovascular Research Advance Access originally published online on January 16, 2009
Cardiovascular Research 2009 81(4):633-634; doi:10.1093/cvr/cvp025
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2009. For permissions please email: journals.permissions@oxfordjournals.org

Carotid body and sympathetic activation in heart failure: a story of sensors and sensitivity

Río Aguilar Torres

Servicio de Cardiología, Hospital Universitario Vall d'Hebron, P° Vall d'Hebron 119–129, Barcelona 08035, Spain

Corresponding author. Tel: +34 93 2746212; fax: +34 93 2746244. E-mail address: rioaguilar@pulso.com

This editorial refers to ‘Role of CuZn superoxide dismutase on carotid body function in heart failure rabbits’ by Yanfeng Ding et al.,8 pp. 678–685, this issue.

The first 10% of the full text of this article appears below.

The carotid body (CB) is a peripheral chemoreceptor organ that contains clusters of electrically excitable secretory cells, the glomus cells, which express several types of membrane ion channels that influence its excitability. These cells act as sensors or chemotransducers, detecting different chemical stimuli and triggering an action potential in the afferent fibres that lie in synaptic apposition. The main stimulus for glomus cells are constant falls in arterial partial pressure of oxygen (PaO2), in opposition to the highly sensitive partial pressure of carbon dioxide (PaCO2) chemoreceptors at the central nervous system (CNS). The afferent nerve supply of the CB, which is accompanied by the afferent fibres from carotid sinus . . . [Full Text of this Article]


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Related Article

Role of CuZn superoxide dismutase on carotid body function in heart failure rabbits
Yanfeng Ding, Yu-Long Li, Matthew C. Zimmerman, Robin L. Davisson, and Harold D. Schultz
Cardiovasc Res 2009 81: 678-685. [Abstract] [Full Text] [PDF]