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Cardiovascular Research Advance Access originally published online on August 27, 2009
Cardiovascular Research 2009 84(2):180-181; doi:10.1093/cvr/cvp299
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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author 2009. For permissions please email: journals.permissions@oxfordjournals.org.

Duration of heart failure and the risk of atrial fibrillation: different mechanisms at different times?

Andrew C. Rankin* and Antony J. Workman

BHF Glasgow Cardiovascular Research Centre, University of Glasgow, 126 University Place, Glasgow G12 8TA, UK

* Corresponding author. Tel: +44 141 211 4833; fax: +44 141 552 4683; E-mail address: acr1a@clinmed.gla.ac.uk

This editorial refers to ‘Chronic heart failure and the substrate for atrial fibrillation’ by A. Sridhar et al., pp. 227–236, this issue.

The first 10% of the full text of this article appears below.

Chronic heart failure increases the risk of atrial fibrillation (AF), with the prevalence of AF paralleling the severity of heart failure.1 Factors that underlie this increased susceptibility to AF may include electrical, structural, and neurohumoral changes.2 In AF, it is recognized that atrial electrophysiological remodelling occurs and contributes to the perpetuation of the arrhythmia, most notably the decrease of effective refractory period (ERP) which predisposes to re-entry by shortening the wavelength. Does heart failure cause similar changes in atrial electrophysiology that predispose to the arrhythmia?

An extensively studied dog model, in which heart failure was induced by rapid ventricular pacing, has suggested not: atrial electrophysiological changes . . . [Full Text of this Article]


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Chronic heart failure and the substrate for atrial fibrillation
Arun Sridhar, Yoshinori Nishijima, Dmitry Terentyev, Mahmood Khan, Radmila Terentyeva, Robert L. Hamlin, Tomohiro Nakayama, Sandor Gyorke, Arturo J. Cardounel, and Cynthia A. Carnes
Cardiovasc Res 2009 84: 227-236. [Abstract] [Full Text] [PDF]