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Cardiovascular Research 2007 76(1):3-4; doi:10.1016/j.cardiores.2007.07.007
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Copyright © 2007, European Society of Cardiology

Sphingosine kinase isoforms and cardiac protection

Yasuhiro Nishino, Ian Webb and Michael S. Marber*

The Cardiovascular Division, Kings College London, The Rayne Institute, St Thomas' Hospital, London, UK

*Corresponding author. Department of Cardiology, Kings College London, The Rayne Institute, St Thomas' Hospital, London SE1 7EH, UK mike.marber@kcl.ac.uk

Received 4 July 2007; accepted 17 July 2007

The first 10% of the full text of this article appears below.

See article by Jin et al. [3] (pages 41–50) in this issue.

Sphingolipids are complex, ubiquitous lipids commonly believed to protect the cell surface against harmful environmental factors by forming a mechanically stable and chemically resistant outer leaflet of the plasma membrane lipid bilayer. Over the last decade relatively simple sphingolipid catabolites, such as ceramide and sphingosine-1-phosphate (S1P), have been identified as signal-transducing molecules involved in the regulation of cell growth, viability, mitosis, cytoskeletal rearrangement, immunity and angiogenesis in a variety of cells and tissues [1]. Ceramide and S1P are products of the membrane phospholipid sphingomyelin, and . . . [Full Text of this Article]


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