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Cardiovascular Research 2007 74(3):337-338; doi:10.1016/j.cardiores.2007.04.002
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Copyright © 2007, European Society of Cardiology

DHEA treatment of pulmonary hypertension: New insights into a complex mechanism

Eric Dubuis

University of Liverpool, School of Clinical Sciences, The Duncan Building, Daulby Street, L69 3GA, Liverpool, UK

dubuis_e@yahoo.fr

Received 1 April 2007; accepted 3 April 2007

The first 10% of the full text of this article appears below.

See article by Oka et al. [5] (pages 377–387) in this issue.

Pulmonary arterial hypertension (PAHT) is an implacable killer. Patients developing PAHT present a survival rate of 40% after 36 months [1]. This deadly pathology remains actually an orphan in terms of long-term treatment and affects more than 100,000 patients in the United States and Europe, with an annual incidence estimated at 2 new cases for primary PAHT and 8 new cases for secondary PAHT per million individuals per year.

Pulmonary hypertension is classified into two main categories. The causes of primary PAHT remain largely . . . [Full Text of this Article]


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