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Cardiovascular Research 2007 73(3):446-447; doi:10.1016/j.cardiores.2006.12.008
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Copyright © 2006, European Society of Cardiology

Triggering of cardiac preconditioning through Na+/K+-ATPase

Javier Inserte*

Servicio de Cardiologia, Hospital Universitari Vall d'Hebron, Passeig Vall d'Hebron, 119-129, 08035 Barcelona, Spain

* Tel.: +34 93 4894038; fax: +34 93 4894032. Email address: jinserte@ir.vhebron.net

Received 5 December 2006; accepted 7 December 2006

The first 10% of the full text of this article appears below.

See article by Pierre et al. [6] (pages 488–496) in this issue.

It has been two decades since the first reports describing the phenomenon of ischemic preconditioning (IP). Data generated during these years describe a wide range of pharmacological triggers of IP, different intracellular signalling pathways, and some end-effectors of the powerful protection that IP exerts against cell death [1].

One of the most recent mechanisms that has been proposed to explain the protection induced by IP involves Na+/K+-ATPase. It is well accepted that the kinetics of Na+ recovery during reperfusion determines the . . . [Full Text of this Article]


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