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Cardiovascular Research 2006 72(2):193-195; doi:10.1016/j.cardiores.2006.08.016
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Copyright © 2006, European Society of Cardiology

Sports or statins for atheroprotection? New insight from Kruppel-like factor 2

Thomas Thuma,b,* and Johann Bauersachsa

aUniversität Würzburg, Universitätsklinikum, Medizinische Klinik und Poliklinik I, Kardiologie, Würzburg, Germany
bUniversität Würzburg, Interdisciplinary Center for Clinical Research (IZKF), Junior Research Group "Cardiac Wounding/Healing", Würzburg, Germany

* Corresponding author. Medizinische Klinik und Poliklinik I, Kardiologie Universitätsklinikum, Bayerische Julius-Maximilians-Universität Josef-Schneider Str. 2 97080 Würzburg Germany. Tel.: +49 931 201 36455; fax: +49 931 201 36664. Email address: Thum_T@klinik.uni-wuerzburg.de

Received 24 July 2006; accepted 24 August 2006

The first 10% of the full text of this article appears below.

See article by van Thienen et al. [6] (pages 231–240) in this issue.

Nitric oxide (NO) plays a central role in the control of vascular homeostasis. Adequate NO production stimulated by continuous laminar flow on the endothelial surface, so-called shear stress, prevents endothelial inflammation and development of endothelial dysfunction. Recently, a novel class of mechanosensitive transcription factors has been identified that in endothelial cells transfer shear stress to various downstream targets such as the endothelial NO synthase (eNOS) [1]. These Kruppel-like factors (KLFs) are zinc finger transcription factors that act as transcriptional activators . . . [Full Text of this Article]


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