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Cardiovascular Research 2006 71(4):618-619; doi:10.1016/j.cardiores.2006.07.003
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Copyright © 2006, European Society of Cardiology

Hypoxia in lung vascular biology and disease

Norbert Weissmann, Friedrich Grimminger and Werner Seegera,*

aUniversity of Giessen Lung Center (UGLC), Medical Clinic II and V, Justus-Liebig-University Giessen, Klinikstrasse 36, 35392 Giessen, Germany

* Corresponding author. Tel.: +49 641 99 42351. Email address: werner.seeger@uglc.de werner.seeger@innere.med.uni-giessen.de

Received 3 July 2006; accepted 4 July 2006

The first 10% of the full text of this article appears below.

See reviews in the series by Weissmann et al. [3] (pages 620–629), Weir and Olschewski [4] (pages 630–641), and Fandrey et al. [6] (pages 642–651) in this issue and by Ghohani et al. [7] and Maggiorini [8] in next month's issue.

With the evolution of photosynthesis, organisms had to cope with a dramatic environmental change: the presence of oxygen. Despite the initial toxicity of this gas, the presence of molecular oxygen offered a more effective energy metabolism. As a result, animals and humans require oxygen to survive, and a shortage of oxygen, hypoxia, can evoke life-threatening conditions. To maintain an optimal energy supply for the body, the lung is the first-line organ . . . [Full Text of this Article]


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