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Cardiovascular Research 2006 70(2):167-169; doi:10.1016/j.cardiores.2006.03.009
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Copyright © 2006, European Society of Cardiology

Bringing preconditioning and postconditioning into focus

David Garcia-Doradoa,*, Jacob Vinten-Johansenb and Hans Michael Piperc

aHospital Univ Vall d'Hebron, Laboratorio de Cardiología Experimental, Servicio de Cardiología, Pg. Vall d'Hebron 119-12908035 Barcelona, Spain
bEmory University School of Medicine, Carlyle Fraser Heart Center, Atlanta, GA, USA
cPhysiologisches Institut, Justus Liebig University of Giessen, Giessen, Germany

* Corresponding author. Tel.: +34 93 489 4038; fax: +34 93 489 4032. Email address: dgdorado@vhebron.net

Received 7 March 2006; accepted 14 March 2006

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After many years of debate during which the preponderance of opinion was skeptical about the experimental and clinical existence of reperfusion injury (with attitudes ranging between polite condescendence and overt denial), it can be said that the controversy has been now largely resolved, and that the death of cardiomyocytes during reperfusion is now generally admitted as a fact. Perhaps this agreement is still more firm in the experimental literature than in clinical cardiology. The discovery of the phenomenon of postconditioning [1] has undoubtedly contributed to this change of opinion. The first reports of postconditioning the human heart by Laskey et al. and Ovize et al. certainly support the concept of reperfusion injury as a clinical entity that can be measured even in a small group of patients [2,3]. The report by Zhao et . . . [Full Text of this Article]


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