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Cardiovascular Research 2006 70(1):3-5; doi:10.1016/j.cardiores.2006.01.006
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Copyright © 2006, European Society of Cardiology

Targeting bone marrow to treat vascular diseases: Accelerated vascular healing by colony stimulating factor

Julie Sainza and Masataka Sataa,b,*

aDepartment of Cardiovascular Medicine, University of Tokyo, Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo 113-8655, Japan
bDepartment of Advanced Clinical Science and Therapeutics, University of Tokyo Graduate School of Medicine, Tokyo 113-8655, Japan

* Corresponding author. Department of Cardiovascular Medicine, University of Tokyo, Graduate School of Medicine, 7-3-1 Hongo, Bunkyo-ku, Tokyo 113-8655, Japan. Tel.: +81 3 3815 5411; fax: +81 3 3814 0021. Email address: msata-circ@umin.ac.jp

Received 31 December 2005; accepted 9 January 2006

The first 10% of the full text of this article appears below.

See article by Yoshioka et al. [6] (pages 61–69) in this issue.

It is a generally accepted view that endothelial damage triggers the pathogenesis of various types of vascular diseases [1]. A growing body of evidence suggests that bone marrow (BM)-derived cells may participate in arterial repair after injury [2,3] by homing to the injured vessel and differentiating into endothelial cells (ECs). Walter et al. suggested that the mobilization of endothelial progenitor cells (EPCs) from BM after vascular injury may mediate accelerated reendothelialization and reduced neointima formation by statin therapy [4]. It was reported that granulocyte colony-stimulating factor (G-CSF), a major regulator of haemopoiesis and the innate immune system, potently . . . [Full Text of this Article]


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