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Cardiovascular Research 2006 69(1):7-8; doi:10.1016/j.cardiores.2005.10.009
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Copyright © 2005, European Society of Cardiology

NF{kappa}B, heat shock proteins, HSF-1, and inflammation

A.A. Knowlton*

Molecular and Cellular Cardiology, Department of Medicine, University of California, Davis, Davis, CA and Sacramento VA Medical Center, United States

* Molecular and Cellular Cardiology, Genomics and Biomedical Sciences Facility, Rm 6317, University of California, Davis, 451 East Health Sciences Way, Davis, CA 95616, United States. Tel.: +1 530 752 5461; fax: +1 530 754 7167. Email address: aaknowlton@ucdavis.edu

Received 26 September 2005; revised 18 October 2005; accepted 25 October 2005

The first 10% of the full text of this article appears below.

See article by Y. Chen and R.W. Currie [1] (pages 66–75) in this issue.

Investigators are beginning to unravel the intricate relationship between NF{kappa}B, heat shock proteins (HSPs), and heat shock factor (HSF)-1. New information is added to this complex picture with the novel finding that HSF-1 modulates AP-1 activation, adding yet another layer of complexity [1]. HSF-1, a transcription factor for the heat shock proteins, is a key component of the heat shock response. The activation of HSF-1 . . . [Full Text of this Article]


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