Skip Navigation

Cardiovascular Research 2005 68(3):353-354; doi:10.1016/j.cardiores.2005.09.009
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Li, D.
Right arrow Articles by Mehta, J. L.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Li, D.
Right arrow Articles by Mehta, J. L.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2005, European Society of Cardiology

Oxidized LDL, a critical factor in atherogenesis

Dayuan Li* and Jawahar L. Mehta

Department of Medicine, University of Arkansas for Medical Sciences and Central Arkansas Veterans Healthcare System, Little Rock, AR, United States

* Corresponding author. Department of Internal Medicine, University of Arkansas for Medical Sciences, 4301 W Markham, Slot 532, Little Rock, AR 72205, United States. Tel.: +1 501 686 6724. Email address: lidayuan@uams.edu

Received 2 September 2005; accepted 20 September 2005

The first 10% of the full text of this article appears below.

See also article by Zhu et al. [3] (pages 425–432) in this issue.

There is a great deal of experimental, epidemiological, and clinical evidence suggesting that disorders of lipid metabolism play an important role in the pathogenesis of atherosclerosis. To date, there have been many studies investigating the mechanisms by which high levels of LDL-cholesterol affect biology of vessels and cause atherosclerotic lesion formation [1,2]. It has become abundantly clear over the last decade that the oxidatively modified form of LDL (ox-LDL) is more important than native LDL in atherogenesis.

The study in this . . . [Full Text of this Article]


   Oxidized LDL and endothelial dysfunction/injury
 

   Oxidized LDL and its receptor LOX-1 in endothelium
 

   Ox-LDL and atherosclerosis
 

Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 J. Lipid Res.Home page
P. Martinez-Miguel, V. Raoch, C. Zaragoza, J. M. Valdivielso, M. Rodriguez-Puyol, D. Rodriguez-Puyol, and S. Lopez-Ongil
Endothelin-converting enzyme-1 increases in atherosclerotic mice: potential role of oxidized low density lipoproteins
J. Lipid Res., March 1, 2009; 50(3): 364 - 375.
[Abstract] [Full Text] [PDF]

Home page
 J. Biol. Chem.Home page
I. M. Qahwash, A. Boire, J. Lanning, T. Krausz, P. Pytel, and S. C. Meredith
Site-specific Effects of Peptide Lipidation on -Amyloid Aggregation and Cytotoxicity
J. Biol. Chem., December 21, 2007; 282(51): 36987 - 36997.
[Abstract] [Full Text] [PDF]