Skip Navigation

Cardiovascular Research 2005 68(3):350-352; doi:10.1016/j.cardiores.2005.09.008
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Fischer, J.W.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Fischer, J.W.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2005, European Society of Cardiology

Dexamethasone: Effects on neointimal hyperplasia and vessel integrity

J.W. Fischer*

Molekulare Pharmakologie, Institut für Pharmakologie und Klinische Pharmakologie, Heinrich Heine Universität Düsseldorf, Moorenstrasse 5, D-40225 Düsseldorf, Germany

* Tel.: +49 211 81 12513x12500; fax: +49 211 81 14781. Email address: jens.fischer@uni-duesseldorf.de

Received 15 August 2005; accepted 20 September 2005

The first 10% of the full text of this article appears below.

See article by Pires et al. [11] (pages 415–424) in this issue.

The clinical success of balloon angioplasty is limited by restenosis, which occurs in up to 50% of patients. Lumen compromise and restenosis are caused by acute elastic recoil and late constrictive remodeling and to a lesser degree by neointimal hyperplasia [1,2]. The application of bare metal stents (BMS) has revolutionized percutaneous coronary interventions by reducing the rate of clinical complications and the need for target vessel revascularization. Although BMS mechanically prevent the decrease of total vessel area caused by constrictive remodeling, in-stent restenosis still reduces the clinical success rate of BMS substantially. In contrast to restenosis after balloon angioplasty, after stent implantation restenosis is caused exclusively by ingrowth of a thickened neointima as the . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 BloodHome page
D. C. Johnson, S. Corthals, C. Ramos, A. Hoering, K. Cocks, N. J. Dickens, J. Haessler, H. Goldschmidt, J. A. Child, S. E. Bell, et al.
Genetic associations with thalidomide mediated venous thrombotic events in myeloma identified using targeted genotyping
Blood, December 15, 2008; 112(13): 4924 - 4934.
[Abstract] [Full Text] [PDF]

Home page
 Cardiovasc ResHome page
Y. D. Krom, N. M.M. Pires, J. W. Jukema, M. R. de Vries, R. R. Frants, L. M. Havekes, K. W. van Dijk, and P. H.A. Quax
Inhibition of neointima formation by local delivery of estrogen receptor alpha and beta specific agonists
Cardiovasc Res, January 1, 2007; 73(1): 217 - 226.
[Abstract] [Full Text] [PDF]