Skip Navigation

Cardiovascular Research 2005 67(2):182-183; doi:10.1016/j.cardiores.2005.05.012
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Ventura, C.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Ventura, C.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2005, European Society of Cardiology

Forced myocardin expression primes cardiac and smooth muscle transcription patterning in human mesenchymal stem cells

Carlo Ventura*

Laboratory of Molecular Biology and Stem Cell Engineering, National Institute of Biostructures and Biosystems, University of Bologna, 40138 Bologna, Italy

* Present address: S. Orsola-Malpighi Hospital, Institute of Cardiology, Via Massarenti N.g, 40138 Bologna, Italy. Tel.: +39 051 6363605; fax: +39 051 344859. Email address: cvent@libero.it

Received 9 May 2005; accepted 10 May 2005

The first 10% of the full text of this article appears below.

See article by van Tuyn et al. [7] (pages 245–255) in this issue.

Loss of cardiomyocytes during myocardial infarction or hereditary cardiomyopathies may represent a major determinant in the progression toward heart failure. The reported ability of haematopoietic bone marrow cells to afford myocardial regeneration after direct injection in experimental animals [1] has triggered a number of clinical trials in humans. Nevertheless, the initial observations and the scientific underpinning of the human trials have been challenged by a number of studies using a foreign gene, LacZ or GFP, to . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
Stem CellsHome page
R. Madonna, J. T. Willerson, and Y.-J. Geng
Myocardin A Enhances Telomerase Activities in Adipose Tissue Mesenchymal Cells and Embryonic Stem Cells Undergoing Cardiovascular Myogenic Differentiation
Stem Cells, January 1, 2008; 26(1): 202 - 211.
[Abstract] [Full Text] [PDF]


Home page
J. Cell Sci.Home page
E. S. Jeon, H. J. Moon, M. J. Lee, H. Y. Song, Y. M. Kim, Y. C. Bae, J. S. Jung, and J. H. Kim
Sphingosylphosphorylcholine induces differentiation of human mesenchymal stem cells into smooth-muscle-like cells through a TGF-{beta}-dependent mechanism
J. Cell Sci., December 1, 2006; 119(23): 4994 - 5005.
[Abstract] [Full Text] [PDF]


Home page
Am. J. Physiol. Cell Physiol.Home page
B. P. Herring, O. El-Mounayri, P. J. Gallagher, F. Yin, and J. Zhou
Regulation of myosin light chain kinase and telokin expression in smooth muscle tissues
Am J Physiol Cell Physiol, November 1, 2006; 291(5): C817 - C827.
[Abstract] [Full Text] [PDF]