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Cardiovascular Research 2005 65(4):768-769; doi:10.1016/j.cardiores.2004.12.011
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Copyright © 2004, European Society of Cardiology

Tetrahydrobiopterin and eNOS dimer/monomer ratio–a clue to eNOS uncoupling in diabetes?

Johann Bauersachs* and Andreas Schäfer

Medizinische Klinik der Julius-Maximilians-Universität Würzburg, Germany

* Corresponding author. Medizinische Universitätsklinik, Josef-Schneider-Str. 2, D-97080 Würzburg, Germany. Tel.: +49 931 201 1; fax: +49 931 201 36302. Email address: j.bauersachs@medizin.uni-wuerzburg.de

Received 9 December 2004; accepted 21 December 2004

The first 10% of the full text of this article appears below.

See article by Cai et al. [14] (pages 823–831) in this issue.

Nitric oxide (NO) generated by the endothelial NO synthase (eNOS) critically determines vascular tone as well as vascular wall homeostasis [1]. Reduced NO bioavailability, which is associated with an increase in the formation of reactive oxygen species within the vascular wall, is the key determinant of endothelial dysfunction. The paradoxical finding of . . . [Full Text of this Article]


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