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Cardiovascular Research 2004 64(3):377-378; doi:10.1016/j.cardiores.2004.09.011
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

Focal adhesion kinase and angiogenesis. Where do we go from here?

Maria Teresa Rizzo*

Signal Transduction Laboratory, Methodist Research Institute, 1701 N. Senate, Rm. E504, Indianapolis, IN 46202, USA
Department of Pharmacology, Indiana University School of Medicine, Indianapolis, IN, USA

* Signal Transduction Laboratory, Methodist Research Institute, 1701 N. Senate, Rm. E504, Indianapolis, IN 46202, USA. Tel.: +1 317 962 6891; fax: +1 317 962 9369. Email address: mrizzo@clarian.org

Received 13 September 2004; accepted 15 September 2004

The first 10% of the full text of this article appears below.

See article by Peng et al. (pages 421–430) in this issue.

Processes involved in the generation of new blood vessels during embryonic development, wound healing, ischemia, inflammation and tumor growth have captured the imagination of investigators for many years. Numerous studies have led to the well-founded expectation that regulation of angiogenesis will soon be a therapeutically useful approach for a wide range of disorders, ranging from inflammation to neoplastic growth [1]. The angiogenic response is a multifaceted and highly orchestrated process initiated by the endothelial cells of preexisting capillaries. [2]. Responding to a wide array of external stimuli, endothelial cells engage in a series of . . . [Full Text of this Article]


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