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Cardiovascular Research 2004 63(4):577-579; doi:10.1016/j.cardiores.2004.06.017
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

Mechanical influence of cyclic stretch on vascular endothelial cells

Patrick Lacolley*

INSERM EMI 0107, 15, rue de l'Ecole de Médecine, 75270 Paris cedex 06, France

* Tel.: +33 1 44 07 90 38; fax: +33 1 44 07 90 40. Email address: lacolley@ccr.jussieu.fr

Received 5 June 2004; accepted 21 June 2004

The first 10% of the full text of this article appears below.

See article by Von Offenberg Sweeney et al. [3] (pages 625–634) in this issue.

Most of the biological phenomena acting on arterial vessels are non-linear. The biological response of cells to mechanical stimuli differs substantially depending on whether they are exposed to static or pulsatile stress [1]. Since the first work by Glagov and his group [2], the role of cyclic hemodynamic forces has been emphasized. In the present issue, Von Offenberg Sweeney et al. [3] have studied the role of cyclic strain on the endothelial matrix metalloproteinase-2 expression and activity. They have also shown the importance of metalloproteinases within the frame of vascular remodeling and its consequences in clinical situations.

Endothelial cells (ECs) are mainly exposed to blood shear stress in the direction of . . . [Full Text of this Article]


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