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Cardiovascular Research 2004 61(2):206-207; doi:10.1016/j.cardiores.2003.12.001
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

Transgenic models in cardiac arrhythmias: how close can we get to the bedside?

Carlo Napolitano*

Molecular Cardiology Laboratories, IRCCS Fondazione S Maugeri, Via Ferrata 8, 27100 Pavia, PV, Italy

* Tel.: +39-382-592050; fax: +39-382-592094. cnapolitano@fsm.it

Received 30 November 2003; accepted 2 December 2003

The first 10% of the full text of this article appears below.

See article by Tian et al. [8] (pages 256–267) in this issue.

The exponential growth of studies exploring the molecular bases of arrhythmogenesis is one of the most relevant trends recently taking place in the area of cardiovascular pathophysiology. The starting point of this line of research was the discovery of the DNA coding abnormalities causing Mendelian diseases (such as the Long QT syndrome (LQTS), the Brugada syndrome and catecholaminergic polymorphic ventricular tachycardia) that alter cardiac excitability and cause sudden cardiac death. This step constituted the inception of . . . [Full Text of this Article]


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