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Cardiovascular Research 2004 61(1):196-197; doi:10.1016/j.cardiores.2003.10.020
© 2004 by European Society of Cardiology
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Copyright © 2004, European Society of Cardiology

Activity of the Akt/GSK-3β pathway in the failing human heart before and after left ventricular assist device support

Peter Razeghi and Heinrich Taegtmeyer*

Department of Internal Medicine, Division of Cardiology, The University of Texas-Houston Medical School, 6431 Fannin, MSB 1.246, Houston, TX 77030, USA

* Corresponding author. Tel.: +1-713-500-6569; fax: +1-713-500-6556. heinrich.taegtmeyer@uth.tmc.edu

Received 14 August 2003; accepted 23 October 2003

The first 10% of the full text of this article appears below.

We wish to comment on the report by Baba et al. [1] The authors measured the activity of several kinases that are involved in the regulation of hypertrophy and apoptosis in the failing human heart before and after mechanical unloading with a left ventricular assist device (LVAD) [1]. They found that the activity of Erks and Akt in the failing human heart decreased after LVAD support, while that of glycogen synthase kinase-3β . . . [Full Text of this Article]


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