Skip Navigation

Cardiovascular Research 2003 60(3):463-464; doi:10.1016/j.cardiores.2003.10.015
© 2003 by European Society of Cardiology
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Kaplinsky, E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Kaplinsky, E.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2003, European Society of Cardiology

Do we understand why regression of left ventricular hypertrophy is beneficial?

Elieser Kaplinsky

Tel Aviv University, Israel

Received 13 October 2003; revised 13 October 2003; accepted 13 October 2003

The first 10% of the full text of this article appears below.

See article by Botchway et al. [12] (pages 510–517) in this issue.

Hypertrophy is the mechanism by which muscle, any muscle, copes with persistent and repetitive overloads. Any increase in cardiac chamber radius (r) and/or pressure (p) will result in a proportional increase in total wall tension (T) according to the ideal Laplace formula, T = pr. The true load on the individual myocardial fibers can be appreciated by dividing the tension by the width of the chamber wall (h): Ts=(pr)/h. . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?