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Cardiovascular Research 2002 56(3):350-352; doi:10.1016/S0008-6363(02)00672-7
© 2002 by European Society of Cardiology
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Copyright © 2002, European Society of Cardiology

A little gap junctional uncoupling too much

Joris R. de Groot*

Academic Medical Center, Department of Clinical and Experimental Cardiology, Experimental and Molecular Cardiology Group, P.O. Box 22700, 1100 DE Amsterdam, The Netherlands

* Tel.: +31-20-566-3266; fax: +31-20-697-5458 j.r.degroot@amc.uva.nl

Received 17 September 2002; accepted 17 September 2002

The first 10% of the full text of this article appears below.

See article by Pollard et al. [15] (pages 381–392) in this issue.

Ventricular fibrillation (VF) in the setting of acute myocardial ischemia and infarction remains an enormous burden for society, with ~300,000 deaths each year in the United States alone [1,2]. The chance of surviving an event of ischemia-induced VF is inversely proportional to the time elapsing until defibrillation is applied [3]. Therefore, automatic external defibrillators are now more and more being introduced in public areas. However, the vast majority of cases of sudden death occur at home [4].

Therefore, despite the numerous number of studies published in recent decades, there is still a need for better understanding of the mechanism of ischemia-induced arrhythmias. The information that is available to date concerns . . . [Full Text of this Article]


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