Skip Navigation

Cardiovascular Research 2002 56(2):178-180; doi:10.1016/S0008-6363(02)00652-1
© 2002 by European Society of Cardiology
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow E-letters: Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when E-letters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by van der Wal, A. C
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by van der Wal, A. C
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Copyright © 2002, European Society of Cardiology

Chlamydia pneumoniae inside the atherosclerotic plaque—does it affect plaque inflammation and plaque progression?

Allard C van der Wal*

Department of Cardiovascular Pathology, Academic Medical Centre, Meibergdreef 9, 1105 AZ Amsterdam, The Netherlands

* Tel.: +31-20-566-5633; fax: +31-20-691-4738. a.c.vanderwal@amc.uva.nl

Received 27 August 2002; accepted 28 August 2002

The first 10% of the full text of this article appears below.

See article by Ezzahiri et al. [5] (pages 269–276) in this issue.


   1. Introduction
 
Exposure to Chlamydia pneumoniae (Cp) is very common among the human population world-wide. Circa 50% of adults bear antibodies to Cp and re-infections occur frequently: most people have 2–3 infections in a lifetime [1]. In Cardiovascular Medicine, interest for such a common opportunistic agent was initiated by insights in seroepidemiologic associations between Cp infection and the risk of atherosclerotic vascular disease, myocardial infarction and stroke [2]. Atherosclerosis has recently been classified as a chronic inflammatory disease [3], so potentially the disease could indeed be initiated or modulated by infectious agents. Still, despite numerous clinical and pathological observations . . . [Full Text of this Article]


   2. Chlamydia pneumoniae infection and plaque inflammation
 

   3. Chlamydia pneumoniae infection and plaque progression
 

Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Cardiovasc ResHome page
M. D. de Kruif, E. C.M. van Gorp, T. T. Keller, J. M. Ossewaarde, and H. ten Cate
Chlamydia pneumoniae infections in mouse models: relevance for atherosclerosis research
Cardiovasc Res, February 1, 2005; 65(2): 317 - 327.
[Abstract] [Full Text] [PDF]