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Cardiovascular Research 2002 53(4):779-781; doi:10.1016/S0008-6363(02)00235-3
© 2002 by European Society of Cardiology
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Copyright © 2002, European Society of Cardiology

Vitronectin is implicated as the matrix takes control of neointima formation

Andrew C. Newby*

Bristol Heart Institute, University of Bristol, Bristol Royal Infirmary, Level 7, Bristol BS2 8HW, UK

* Tel.: +44-117-928-3582; fax: +44-117-928-3581 a.newby@bris.ac.uk

Received 28 December 2001; accepted 28 December 2001

KEYWORDS Extracellular matrix; Artherosclerosis

The first 10% of the full text of this article appears below.

See article by Dufourcq et al. [9] (pages 953–963) in this issue.

The normal blood vessel wall contains two functionally distinct types of vascular extracellular matrix, namely basement membranes and interstitial matrix. A basement membrane consisting of type IV collagen, the multi-adhesive glycoprotein, laminin, and a variety of heparan sulphate proteoglycans, surrounds all quiescent, contractile smooth muscle cells (SMCs). Basement membranes maintain the contractile phenotype, since plating freshly isolated SMCs onto type IV collagen or laminin retards transition to the activated, synthetic phenotype characteristic of vascular repair [1]. . . . [Full Text of this Article]


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