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Cardiovascular Research 2001 51(1):1-3; doi:10.1016/S0008-6363(01)00332-7
© 2001 by European Society of Cardiology
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Copyright © 2000, European Society of Cardiology

Multiple interactions determine cellular electrical processes in the multicellular tissue

Yoram Rudy*

Cardiac Bioelectricity Research and Training Center, 509 Wickenden Building, Case Western Reserve University, Cleveland, OH 44106-7207, USA

yxr@po.cwru.edu

* Tel.: +1-216-368-4051; fax: +1-216-368-8672

Received 19 April 2001;
The first 10% of the full text of this article appears below.

See article by Verkerk et al. [5] (pages 30–40) in this issue.

"Things should be made as simple as possible, but not any simpler" Albert Einstein

The traditional classification of cardiac arrhythmias makes a distinction between focal mechanisms due to abnormal electrophysiological functioning of single cells, and propagation-related mechanisms due to abnormal conduction in the multicellular tissue [1]. Single-cell arrhythmogenecity is associated with triggered activity and after-depolarizations, which are subdivided into early (EAD) and delayed (DAD) afterdepolarizations based on their timing relative to the action potential (AP) [2]. Propagation-based arrhythmias include various types of reentry [3] and spiral-wave activity [4] that involve many interconnected cardiac cells and can occur on different spatial scales (e.g. micro- or macro-reentry). The . . . [Full Text of this Article]


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