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Cardiovascular Research 2001 50(3):423-425; doi:10.1016/S0008-6363(01)00271-1
© 2001 by European Society of Cardiology
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Copyright © 2001, European Society of Cardiology

Transmural repolarization gradients in vivo: the flukes and falls of the endocardium

Marc A. Vos* and Jérôme G.M. Jungschleger

Department of Cardiology, University Hospital Maastricht, Cardiovascular Research Institute Maastricht, P.O. Box 5800, 6202 AZ Maastricht, The Netherlands

* Corresponding author. Tel.: +31-43-387-5097; fax: +31-43-387-5104 m.vos@cardio.azm.nl

Received 26 February 2001; accepted 26 February 2001

The first 10% of the full text of this article appears below.

See article by Taggart et al. [3] (pages 454–462) in this issue.

In the early 90's the Antzelevitch group described the M-cell [1], in which the M originally refers to midmyocardium but has also been used for Masonic or Moe, as an acknowledgement to the people behind the initiative to perform research in Utica, NY. Since its introduction there has been accumulating in-vitro evidence that this cardiac cell type electrophysiologically and pharmacologically differs from (sub-)epi- and (sub-)endocardial cells of the left ventricular free wall.

In vitro studies revealed that the intrinsic differences in morphology and duration of the action potential (APD) of the different cell layers are emphasized under less physiological circumstances, such as bradycardia, sudden rate changes or acquired long QT, which can be induced by either drugs or pathological . . . [Full Text of this Article]


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