Na+/H+ exchange in ischemia, reperfusion and preconditioning

doi:10.1016/S0008-6363(01)00228-0

Cardiovascular Research 2001 50(1):162-163; doi:10.1016/S0008-6363(01)00228-0
© 2001 by European Society of Cardiology

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Na⁺/H⁺ exchange in ischemia, reperfusion and preconditioning

Metin Avkiran^a^,*, Garrett Gross^b, Morris Karmazyn^c, Hermann Klein^d, Elizabeth Murphy^e and Kirsti Ytrehus^f

^aCentre for Cardiovascular Biology and Medicine, King's College London, The Rayne Institute, St. Thomas' Hospital, London, SE1 7EH UK
^bMedical College of Wisconsin, Milwaukee, USA
^cUniversity of Western Ontario, London, Canada
^dStädt Krankenanstalten Idar-Oberstein GmbH, Idar-Oberstein, Germany
^eNIEHS/NIH, Research Triangle Park, USA
^fUniversity of Tromso, Tromso, Norway

^* Corresponding author metin.avkiran@kcl.ac.uk

Received 16 January 2001; accepted 23 January 2001

The first 10% of the full text of this article appears below.

We read with interest the recent paper in Cardiovascular Researchby Xiao and Allen [1], on the role of the cardiac sarcolemmalNa⁺/H⁺ exchanger (NHE) in ‘reperfusion damage’ andischemic preconditioning, and the accompanying editorial byFiolet [2]. The former concludes that (1) NHE is inhibited duringischemia, (2) NHE is activated rapidly during reperfusion andcontributes to reperfusion damage and (3) the protective mechanismof preconditioning is NHE inhibition during early reperfusion.However, the . . . [Full Text of this Article]

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