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Cardiovascular Research 2000 48(1):11-12; doi:10.1016/S0008-6363(00)00188-7
© 2000 by European Society of Cardiology
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Copyright © 2000, European Society of Cardiology

Decrease of delayed rectifier currents in the subacute phase of infarction

Marieke W. Veldkamp*

Department of Experimental Cardiology, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands

* Tel.: +31-20-566-3269; fax: +31-20-697-5458 m.w.veldkamp@amc.uva.nl

Received 1 August 2000; accepted 1 August 2000

The first 10% of the full text of this article appears below.

See article by Jiang et al. [1] (pages 34–43) in this issue.

The paper of Jiang et al. [1] in this issue of Cardiovascular Research – ‘Delayed rectifier K currents have reduced amplitudes and altered kinetics in myocytes from infarcted canine ventricle’ – forms the coping-stone in a long series of investigations into the mechanisms underlying arrhythmias post myocardial infarction (in terms of altered ionic currents). In this paper, delayed rectifier currents were characterized in ventricular myocytes isolated from the surviving layer of epicardium overlying a 5 day-old infarct, the epicardial borderzone (EBZ).


   1 The canine model of myocardial infarction
 
This canine model of myocardial infarction, caused by complete occlusion of the left anterior descending coronary artery, has been thoroughly studied over the . . . [Full Text of this Article]


   2 Pathophysiological significance of IKr/IKs reduction in EBZ
 

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