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Cardiovascular Research 2000 46(1):20-23; doi:10.1016/S0008-6363(00)00014-6
© 2000 by European Society of Cardiology
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Copyright © 2000, European Society of Cardiology

Sex and NO — beyond regulation of vasomotor tone

Matthias Barton*

aCardiology, Department of Medicine, University Hospital, Rämistrasse 100, CH-8091 Zürich, Switzerland

* Tel.: +41-1-255-1111, ext. 163105; fax: +41-1-255-4401 matthiasbarton@compuserve.com

Received 14 January 2000; accepted 17 January 2000

KEYWORDS Atherosclerosis; Contractile function; Myocarditis; Nitric oxide; Reperfusion; Ischemia; Coronary disease

The first 10% of the full text of this article appears below.

See article by Fraser et al. [43] (pages 111–118) in this issue.

The mechanisms contributing to sex differences in susceptibility to and mortality from cardiovascular and renal disease are still unknown. Sexual dimorphisms of coronary artery disease were first suggested by Heberden in 1802 [1], and researchers have described vascular actions of estrogens since the late 19th century [2–4]. Subsequently, several studies provided indirect and direct evidence for an involvement of estrogens in the regulation of vascular tone [5–9]. Most of the mechanisms were only discovered in the past two decades (reviewed in Refs. [10,11]), following the observation by Furchgott and Zawadzki . . . [Full Text of this Article]


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